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학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
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논문 기본 정보
- 자료유형
- 학술대회자료
- 저자정보
- 발행연도
- 2021.4
- 수록면
- 24 - 25 (2page)
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Introduction. In critical limb ischemia (CLI), overproduction of reactive oxygen species (ROS) by mitochondria due to oxygen/nutrient starvation and impairment of neovascularization give rise to muscle damage and limb loss. Cerium oxide nanoparticles (CNP), which considered to regulate ROS, have shown therapeutic utility in various disease models. This study shows that CNP as a pro-angiogenic agent in a mouse hindlimb ischemia model, and reveled the molecular mechanism underlying the pro-angiogenic effect.
Method. After characterization of CNP, a hindlimb ischemia model was established in immunodeficient nude mice by injecting CNP into a ligated region of a femoral artery. Tissue reperfusion and hindlimb salvage were monitored for 3 weeks.
Results. Following CNP administra^_@span style=color:#999999 ^_# ... ^_@/span^_#^_@a href=javascript:; onclick=onClickReadNode('NODE10554012');fn_statistics('Z354','null','null'); style='color:#999999;font-size:14px;text-decoration:underline;' ^_#전체 초록 보기^_@/a^_#
Method. After characterization of CNP, a hindlimb ischemia model was established in immunodeficient nude mice by injecting CNP into a ligated region of a femoral artery. Tissue reperfusion and hindlimb salvage were monitored for 3 weeks.
Results. Following CNP administra^_@span style=color:#999999 ^_# ... ^_@/span^_#^_@a href=javascript:; onclick=onClickReadNode('NODE10554012');fn_statistics('Z354','null','null'); style='color:#999999;font-size:14px;text-decoration:underline;' ^_#전체 초록 보기^_@/a^_#
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UCI(KEPA) : I410-ECN-0101-2021-515-001700345