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논문 기본 정보
- 자료유형
- 학술대회자료
- 저자정보
- 발행연도
- 2021.7
- 수록면
- 98 - 98 (1page)
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Viral infection during pregnancy is correlated with increased frequency of neurodevelopmental disorder, and this is studied in mice prenatally subjected to maternal immune activation (MIA). We previously showed that in pregnancy mice, interleukin-17a (IL-17a) produced by T helper 17 (Th17) cells (CD4+ T helper effector cells involved in multiple inflammatory conditions) induces behavioral and cortical abnormalities in the offspring exposed to MIA. However, it is unclear whether other maternal factors are required to promote MIA-associated phenotypes. Moreover, the underlying mechanisms by which MIA leads to T cell activation with increased IL-17a in the maternal circulation is not well understood. Here we show that MIA phenotypes in offspring require mater^_@span style=color:#999999 ^_# ... ^_@/span^_#^_@a href=javascript:; onclick=onClickReadNode('NODE10617879');fn_statistics('Z354','null','null'); style='color:#999999;font-size:14px;text-decoration:underline;' ^_#전체 초록 보기^_@/a^_#
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