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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2025.2
- 수록면
- 23 - 39 (17page)
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Ferroptosis, an iron-dependent form of regulated cell death, is driven by lipid peroxidation and shaped by metabolic and antioxidant pathways. In immune cells, ferroptosis susceptibility varies by cell types, lipid composition, and metabolic demands, influencing immune responses in cancer, infections, and autoimmune diseases. Therapeutically, targeting ferroptosis holds promise in cancer immunotherapy by enhancing antitumor immunity or inhibiting immunosuppressive cells. This review highlights the metabolic pathways underlying ferroptosis, its regulation in immune cells, its dual role in tumor progression and antitumor immunity, and its context-dependent therapeutic implications for optimizing cancer treatment.
목차
ABSTRACT
INTRODUCTION
FACTORS REGULATING FERROPTOSIS
FERROPTOSIS IN IMMUNE CELLS
FERROPTOSIS IN THE TME AND ITS THERAPEUTIC POTENTIAL
CONCLUSION AND PERSPECTIVE
REFERENCES
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