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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2019.1
- 수록면
- 179 - 186 (8page)
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Background: Recently, the triglyceride glucose (TyG) index has been considered a surrogate marker of insulin resistance which is awell-known pathogenic factor in nonalcoholic fatty liver disease (NAFLD). However, few studies have investigated the relationshipbetween the TyG index and NAFLD. Thus, we investigated the relationship between the TyG index and NAFLD and the effectiveness of the TyG index compared with the homeostasis model assessment of insulin resistance (HOMA-IR) in identifying NAFLD inKorean adults. Methods: Participants of 4,986 who underwent ultrasonography in a health promotion center were enrolled. The TyG index was calculated as ln [fasting triglycerides (mg/dL)×fasting glucose (mg/dL)/2], and HOMA-IR was estimated. NAFLD was diagnosed byultrasonography. Results: Significant differences were observed in metabolic parameters among the quartiles of the TyG index. The prevalence ofNAFLD significantly increased with increment in the TyG index. After adjusting for multiple risk factors, a logistic regression analysis was performed. When the highest and lowest quartiles of the TyG index and HOMA-IR were compared, the odds ratios for theprevalence of NAFLD were 2.94 and 1.93 (95% confidence interval, 2.32 to 3.72 and 1.43 to 2.61; both P for trend <0.01), respectively. According to the receiver operating characteristic analysis, the TyG index was superior to HOMA-IR in predicting NAFLD. Conclusion: The TyG index and prevalence of NAFLD were significantly related and the TyG index was superior to HOMA-IR inpredicting NAFLD in Korean adults.
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참고문헌 (40)
참고문헌 신청
Browning JD / 2004 / Prevalence of hepatic steatosis in an urban population in the United States : impact of ethnicity / Hepatology 40:1387~1395
Smits MM / 2013 / Non-alcoholic fatty liver disease as an independent manifestation of the metabolic syndrome : results of a US national survey in three ethnic groups / J Gastroenterol Hepatol 28:664~670
Simeone JC / 2017 / Clinical course of nonalcoholic fatty liver disease: an assessment of severity, progression, and outcomes / Clin Epidemiol 9:679~688
Ahmed A / 2015 / Nonalcoholic fatty liver disease review: diagnosis, treatment, and outcomes / Clin Gastroenterol Hepatol 13:2062~2070
Yki-Jarvinen H / 2014 / Non-alcoholic fatty liver disease as a cause and a consequence of metabolic syndrome / Lancet Diabetes Endocrinol 2:901~910
Smits MM / 2013 / Non-alcoholic fatty liver disease as an independent manifestation of the metabolic syndrome : results of a US national survey in three ethnic groups / J Gastroenterol Hepatol 28:664~670
Simeone JC / 2017 / Clinical course of nonalcoholic fatty liver disease: an assessment of severity, progression, and outcomes / Clin Epidemiol 9:679~688
Ahmed A / 2015 / Nonalcoholic fatty liver disease review: diagnosis, treatment, and outcomes / Clin Gastroenterol Hepatol 13:2062~2070
Yki-Jarvinen H / 2014 / Non-alcoholic fatty liver disease as a cause and a consequence of metabolic syndrome / Lancet Diabetes Endocrinol 2:901~910
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