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학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
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연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술대회자료
- 저자정보
- 발행연도
- 2021.1
- 수록면
- 87 - 87 (1page)
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The underlying pathophysiology of neuropathic pain remains a contested topic. One of the strong hypotheses is central disinhibition. Central inhibition is mainly accomplished via spinal interneurons and brainstem descending pathways and via classical inhibitory transmitters such as: aminobutyric acid (GABA), glycine, adrenergic, serotonin (5-HT) and enkephalin, but after nerve injury, laminae I of the spinal dorsal horn and Lamina II inhibitory interneurons undergo trans synaptic excitotoxic changes leading to death, and after nerve injury the protein kinase system will be activated, which causes phosphorylation of GABA receptors and can also lead to reduced inhibition of nociceptive information transmission by central inhibitory interneurons, ultimately causing pain. On the other hand, inhibitory interneurons also become inhibited by acidification of the extracellular environment, and as a membrane-bound enzyme ubiquitously involved in ... 전체 초록 보기
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