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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2002.1
- 수록면
- 11 - 171 (161page)
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The expression of Bis (also called Bag-3), a Bcl-2-binding protein, was investigated in the rat kainicacid (KA) model of temporal lobe epilepsy. Westernblot analysis showed a significant increase in theexpression levels of Bis protein in the hippocampusfollowing the systemic administration of KA. Bisimmunoreactivity increased preferentially in the CA1and CA3 regions, as well as in the hilar region of thedentate gyrus. Experiments with double immunofluorescencerevealed that, in KA-administered rats,the cells expressing Bis were GFAP-expressingreactive astrocytes. The increase in Bis immunoreactivitywas accompanied by increased Bcl-2 inreactive astrocytes in the striatum radiatum,whereas Bcl-2 immunoreactivity in pyramidal neuronswas not affected. These results of the coexpressionof Bis and Bcl-2 in reactive astrocytes inthis seizure model suggest that Bis might modulatethe glial reaction under excitotoxic brain injury,probably by interacting with Bcl-2.
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