지원사업
학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
커뮤니티
연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술대회자료
- 저자정보
- 발행연도
- 2016.7
- 수록면
- 303 - 310 (8page)
이용수
초록· 키워드
오류제보하기
Over the years, researchers have investigated to describe the changes in biomarkers of cardiac damage during recovery from strenuous and prolonged exercise events. A triathlon is an endurance sport-event, which includes swimming, cycling and running with various distances. This study was to investigate the effects of triathlon race on cardiac damage biomarkers in triathletes. Fifteen healthy men participated in the study. Based on performance, they were divided into elite athlete group (EG: n= 7) and non-elite athlete group (NEG: n= 8). Participants’ blood samples were obtained during four periods: before, immediately, 2 hours and 7 days after finishing the race. creatine kinase (CK), creatine kinase-myoglobin (CK-MB), myoglobin, and lactate dehydrogenase (LDH) were significantly increased in both groups immediately after, and 2 hours after finishing the race (p<.05). CK, CK-MB, and myoglobin were completely recovered after 7 days (p<0.05). Hematocrit(Hct) was significantly decreased in both groups (p<0.05) 7 days after the race. LDH was significantly decreased in the EG (p<0.05) only 7 days after the race. Homoglobin(Hb) was significantly decreased in the NEG (p<0.05) only 2 hours after the race. Although cardiac troponin T(cTnT) was significantly increased in the EG but not in the NEG 2hours after the race (p<.05), there was no group-by-time interaction. cTnT was completely recovered in both groups 7 days after the race.
In conclusion, although there is exercise-induced skeletal muscle damage shown by increased CK and CK-MB, cTnT was not increased in both groups immediately after the race. But there was the significant increase in cTnT after 2 hours in EG. We can suggest that it was due to the enormous exercise stress for high performance imposed in EG. However, all cardiac damage markers return to normal range within 1 week.
In conclusion, although there is exercise-induced skeletal muscle damage shown by increased CK and CK-MB, cTnT was not increased in both groups immediately after the race. But there was the significant increase in cTnT after 2 hours in EG. We can suggest that it was due to the enormous exercise stress for high performance imposed in EG. However, all cardiac damage markers return to normal range within 1 week.
목차
Introduction
Material and Methods
Results
Discussion
References
Abstract
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UCI(KEPA) : I410-ECN-0101-2017-910-001021705