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학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
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연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2024.6
- 수록면
- 91 - 98 (8page)
- DOI
- 10.17519/apiculture.2024.06.39.2.91
이용수
초록· 키워드
오류제보하기
Honeybees (Apis mellifera) have been threatened by diseases and their numbers have been noticeably declining. Nosemosis, a disease caused by Nosema ceranae, is now a leading threat to honeybees. N. ceranae is a microsporidian that forms a spore before it infects the host, which makes it especially difficult to control compared to other pathogens. Because the awareness of nosemosis risk increased within decades, N. ceranae has been studied as a pathogen. Some studies showed the up/down-regulation of honeybee mRNA after nosemosis, however, there are no definite mRNAs that can be used as markers for the disease. In this study, to better understand the honeybee’s regulation of mRNA expression after infection by N. ceranae and to determine a marker for nosemosis, 48 mRNAs from the honeybee’s metabolic pathway, immune system, hormone, or unknown mRNAs, were collected from honeybee head and gut and were quantified using real-time PCR. We selected 18 candidates with the largest expression changes and confirmed that more genes from the metabolic pathway were regulated after infection than immune-related genes. Also noteworthy is that the tendency to regulate gene expression in the head and gut differed. In conclusion, many genes, including Aminopeptidase1, GB50026, and Trypsin alpha can be reliable markers for nosemosis, as there are huge differences in the number of transcripts according to the body part.
목차
Abstract
INTRODUCTION
METHODS AND MATERIALS
RESULTS
DISCUSSION
LITERATURE CITED
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