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논문 기본 정보

자료유형
학술저널
저자정보
Shin, Ai-Hyang (School of Life Sciences and Biotechnology, College of Natural Sciences, Kyungpook National University) Oh, Chang-Joo (School of Life Sciences and Biotechnology, College of Natural Sciences, Kyungpook National University) Park, Jeen-Woo (School of Life Sciences and Biotechnology, College of Natural Sciences, Kyungpook National University)
저널정보
대한약학회 Archives of pharmacal research : a publication of the Pharmaceutical Society of Korea Archives of pharmacal research : a publication of the Pharmaceutical Society of Korea 제29권 제7호
발행연도
2006.1
수록면
577 - 581 (5page)

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Oxidative mechanisms are thought to have a major role in cataract formation and diabetic complications. Antioxidant enzymes play an essential role in the antioxidant system of the cells that work to maintain low steady-state concentrations of the reactive oxygen species. When HLE-B3 cells, a human lens cell line were exposed to 50-100 mM glucose for 3 days, decrease of viability, inactivation of antioxidant enzymes, and modulation of cellular redox status were observed. Significant increase of cellular oxidative damage reflected by lipid peroxidation and DNA damage were also found. The glycation-mediated inactivation of antioxidant enzymes may result in the perturbation of cellular antioxidant defense mechanisms and subsequently lead to a pro-oxidant condition and may contribute to various pathologies associated with the long term complications of diabetes.

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