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- 대한약학회 대한약학회 학술대회 대한약학회 2003년도 Proceedings of the Convention of the Pharmaceutical Society of Korea Vol.2-2
- 발행연도
- 2003.1
- 수록면
- 701 - 701 (1page)
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A substantial body of evidence indicates that reactive oxygen intermediates (ROIs) are implicated in pathogenesis of diverse human diseases, including cancer, diabetes and neurodegenerative disorders. Oxidative stress induced by ROIs often causes cell death via apoptosis that is regulated by a plenty of functional genes and their protein products. In the present work, we have investigated the role of bcl-2 in protecting against oxidative death induced by hydrogen peroxide in cultured rat pheochromocytoma (PC12) cells. (omitted)
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BCL-2 OR BCL-XL ATTENUATES HYDROGEN PEROXIDE- AND BETA-AMYLOID-INDUCED OXIDATIVE PC12 CELL DEATH
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15-DEoxy-$d^{12,14}$ Prostaglandin $J_2$ Rescues Pc12 Cells From Hydrogen Peroxide-induced Apoptosis Through Upregulation Of Heme Oxygenase-1
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Overexpression of Bcl-2 protects differentiated PC12 cells against beta amyloid- induced apoptosis through inhibition of NF-kB and p38 MAP kinase activation
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BCL-2 ATTENUATES HYDROGEN PEROXIDE- AND BETA-AMAYLOID-INDUCED OXIDATIVE PC12 CELL DEATH VIA NF-KAPPA B ACTIVAION
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