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논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 저널정보
- 대한소아내분비학회 Annals of Pediatirc Endocrinology & Metabolism Annals of Pediatirc Endocrinology & Metabolism 제14권 제2호
- 발행연도
- 2009.1
- 수록면
- 94 - 99 (6page)
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Insulin resistance and diabetes combine to impair mitochondrial oxidative metabolism and cause lipid accumulation in non-adipose tissues such as skeletal muscles and the liver. The thyroid hormone stimulates thermogenesis, mitochondrial biogenesis, and various metabolisms, including gluconeogenesis and fatty-acid oxidation. Therefore, altered thyroid hormone action may induce the mitochondrial phenotype associated with insulin resistance. This review focuses on the correlation between thyroid hormone function and diabetes and the possible mechanisms associated with intracellular thyroid hormone dysfunction due to impaired metabolism.
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참고문헌 (23)
참고문헌 신청
Bianco AC / 2002 / Biochemistry, cellular and molecular biology, and physiological roles of the iodothyronine selenodeiodinases / Endocr Rev 23 : 38 ~ 89
Bianco AC / 2005 / Cellular and structural biology of the deiodinases / Thyroid 15 : 777 ~ 786
Bianco AC / 2006 / Deiodinases: implications of the local control of thyroid hormone action / J Clin Invest 116 : 2571 ~ 2579
Bradley DJ / 1994 / Alpha and beta thyroid hormone receptor (TR) gene expression during auditory neurogenesis: evidence for TRisoform-specific transcriptional regulation in
Christoffolete MA / 2004 / Mice with targeted disruption of the Dio2 gene have cold-induced overexpression of the uncoupling protein 1 gene but fail to increase brown adipos
Bianco AC / 2005 / Cellular and structural biology of the deiodinases / Thyroid 15 : 777 ~ 786
Bianco AC / 2006 / Deiodinases: implications of the local control of thyroid hormone action / J Clin Invest 116 : 2571 ~ 2579
Bradley DJ / 1994 / Alpha and beta thyroid hormone receptor (TR) gene expression during auditory neurogenesis: evidence for TRisoform-specific transcriptional regulation in
Christoffolete MA / 2004 / Mice with targeted disruption of the Dio2 gene have cold-induced overexpression of the uncoupling protein 1 gene but fail to increase brown adipos
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