지원사업
학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
커뮤니티
연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
논문 기본 정보
- 자료유형
- 학술저널
- 저자정보
- 발행연도
- 2012.1
- 수록면
- 16 - 26 (11page)
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초록· 키워드
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Ginseng is one of the most widely used herbal medicines and is reported to have a wide range of therapeutic and pharmacological applications. Ginseng may also be potentially valuable in treating cardiovascular diseases. Research concerning cardiovascular disease is focusing on purifi ed individual ginsenoside constituents of ginseng to reveal specifi c mechanisms instead of using whole ginseng extracts. The most commonly studied ginsenosides are Rb₁, Rg₁, Rg₃, Rh₁, Re, and Rd. The molecular mechanisms and medical applications of ginsenosides in the treatment of cardiovascular disease have attracted much attention and been the subject of numerous publications. Here, we review the current literature on the myriad pharmacological functions and the potential benefi ts of ginseng in this area. In vitro investigations using cell cultures and in vivo animal models have indicated ginseng’s potential cardiovascular benefi ts through diverse mechanisms that include antioxidation, modifying vasomotor function, reducing platelet adhesion, infl uencing ion channels, altering autonomic neurotransmitters release, and improving lipid profi les. Some 40 ginsenosides have been identifi ed. Each may have different effects in pharmacology and mechanisms due to their different chemical structures. This review also summarizes results of relevant clinical trials regarding the cardiovascular effects of ginseng, particularly in the management of hypertension and improving cardiovascular function.
목차
INTRODUCTION
EFFICACY OF REGULATING INTRACELLULARION CHANNELS
EFFICACY OF ADJUSTING BLOOD PRESSURE
EFFICACY OF IMPROVING MYOCARDIAL PROTECTION
EFFICACY OF REGULATING VASCULAR ENDOTHELIAL CELLS
EFFICACY OF REGULATING VASOMOTOR FUNCTIONS
EFFICACY OF IMPROVING BLOOD CIRCULATION
EFFICACY OF ADJUSTING BLOOD LIPID PROFILE
OXIDATION AND GINSENG
EFFICACY OF ANTI-OXIDANT ACTIVITY
CONCLUSION
REFERENCES
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UCI(KEPA) : I410-ECN-0101-2013-524-001261103