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A 78-year-old man on hemodialysis presented to our hospital with erythrocytosis. He had started hemodialysis 4 years previously, with a hemoglobin level of 9.8 g/dL,and was administered erythropoiesis stimulating agents and ferrous sulfate. Twoyears previously, his hemoglobin level increased to 14.5 g/dL and the treatment foranemia was discontinued. He continued hemodialysis thrice weekly; however, thehemoglobin level had increased to 17.0 g/dL at the time of presenting to ourhospital. His serum erythropoietin level was 31.4 mIU/mL (range, 3.7–31.5 mIU/mL),carboxyhemoglobin level was 0.6% (range, 0–1.5%), and oxygen saturation inambient air was 95.4%. The JAK2 V617F mutation was not observed and otherbone marrow abnormalities were not identified. The patient was diagnosed withbladder cancer and a transurethral resection was performed. Eight months afterthe treatment of bladder cancer, his hemoglobin level was 15.1 g/dL, and he wasdiagnosed with idiopathic erythrocytosis.

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