지원사업
학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
커뮤니티
연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
초록· 키워드
오류제보하기
Lance-Adams syndrome (LAS) is a rare neurological disorder that may occur after cardiopulmonary resuscitation. The LAS is usually caused by hypoxic changes. Neuroimaging studies show that the brain pathology of LAS patients is not uniform, and the pathophysiology of the myoclonus can vary from patient to patient. Our case study contributes to this etiological heterogeneity by neuroimaging and transcranial magnetic stimulation (TMS). In patients with rare brain conditions such as LAS, a combination of brain stimulation methods, such as TMS, and diffusion tensor imaging can provide insights into this condition's pathophysiology. These insights can facilitate the development of more effective therapies.
목차
등록된 정보가 없습니다.
참고문헌 (20)
참고문헌 신청
Bouwes A / 2012 / Acute posthypoxic myoclonus after cardiopulmonary resuscitation / BMC Neurol 12:63
Malhotra S / 2012 / Lance-Adams syndrome : difficulties surrounding diagnosis, prognostication, and treatment after cardiac arrest / Anesth Essays Res 6:218~222
Perez CA / 2016 / Cognitive and functional consequence of cardiac arrest / Curr Neurol Neurosci Rep 16:70
Freund B / 2017 / Post-hypoxic myoclonus : differentiating benign and malignant etiologies in diagnosis and prognosis / Clin Neurophysiol Pract 2:98~102
Groppa S / 2012 / A practical guide to diagnostic transcranial magnetic stimulation : report of an IFCN Committee / Clin Neurophysiol 123:858~882
Malhotra S / 2012 / Lance-Adams syndrome : difficulties surrounding diagnosis, prognostication, and treatment after cardiac arrest / Anesth Essays Res 6:218~222
Perez CA / 2016 / Cognitive and functional consequence of cardiac arrest / Curr Neurol Neurosci Rep 16:70
Freund B / 2017 / Post-hypoxic myoclonus : differentiating benign and malignant etiologies in diagnosis and prognosis / Clin Neurophysiol Pract 2:98~102
Groppa S / 2012 / A practical guide to diagnostic transcranial magnetic stimulation : report of an IFCN Committee / Clin Neurophysiol 123:858~882
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