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- 대한약학회 대한약학회 학술대회 대한약학회 2003년도 Proceedings of the Convention of the Pharmaceutical Society of Korea Vol.1
- 발행연도
- 2003.1
- 수록면
- 119 - 120 (2page)
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One of the most frequent defects in human cancer is the uncontrolled activation of the ras-signaling pathways. Elevated p21 ras expression is associated with tumor aggressiveness in breast cancer including the extent of invasion into fat tissues. infiltration into lymphatic vessels and tumor recurrence, We demonstrate that H-ras, but not N-ras, upregulates matrix metalloproteinase(MMP)-2 expression and induces invasive phenotype in MCF10A human breast epithelial cells. (omitted)
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Activation of MKK6 induces invasive and migrative phenotypes in MCF10A human breast epithelial cells
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A Role of p38 MAPK on H-ras-Induced Invasion and Motility in MCF 10A Human Breast Epithelial Cells
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Differential Functions of Ras for Malignant Phenotypic Conversion
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Roles of Phosphatidylinositol 3-Kinase(PI3K) and Rac1
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Transforming Growth Factor-${\beta}$ (TGF-${\beta}$) Induces Invasion and Migration of Ras- Transformed MCF10A Human Breast Epithelial Cells
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Activation of p38 mitogen-activated protein kinase in H-ras MCF 10A cells: Possible ole in H-ras-induced invasive phenotype
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Chemistry and Biology of Ras Farnesyltransferase
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Ras Signaling in Invasion and Motility of MCF10A Human Breast Epitherial Cells
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Retroviral Delivery of TIMP-2 Inhibits H-ras-induced Migration and Invasion in MCF10A Human Breast Epithelial Cells
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Activation of PI3K is not Sufficient but Required for H-ras-induced Invasive Phenotype of MCF10A Cells
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Roles of PI3K and Rac pathways in H-ras induced invasion and motility
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