지원사업
학술연구/단체지원/교육 등 연구자 활동을 지속하도록 DBpia가 지원하고 있어요.
커뮤니티
연구자들이 자신의 연구와 전문성을 널리 알리고, 새로운 협력의 기회를 만들 수 있는 네트워킹 공간이에요.
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Background : Pure motor hemiplegia(PMH) is known as a syndrome of lacunar infarct. This study is designed to investigate other etiologies of the PMH and sites of the responsible lesion. Methods : Of 927 stroke patients who underwent CT or MRI, 261 patients with newly developed PMH were selected. All strokes causing PMH were divided into three types ; lacunar infarct(defined as a deep-seated small infarct < 2cm in diameter), non-lacunar infarct(defined as an infarct anywhere . 2cm in diameter), and intracerebral hemorrhage. Results : Among 261 PMH, 183 had lacunar infarcts(70%), 48 had non-lacunar infarcts(18%) and 30 had intracerebral hemorrhage(12%). The proportion of PMH in each type of stroke was : 183(48%) of 381 lacunar infarcts, 48(12%) of 395 non-lacunar infarcts and 30(20%) of 151 intracerebral hemorrhage. The corona radiata(80/183; 44%) was more frequent responsible lesion site than the striatocapsular area(63/183; 34%). Striatocapsular infarct(22/48; 46%) and superior division - middle cerebral artery infarct(17/48; 35%) were frequently observed among various types of non-lacunar infarcts leading to PMH. Conclusions : Our data show that non-lacunar infarct and ICH occupy a significant portion of PMH. PMH should not always be considered as indicating a lacunar infarct. Korean Journal of Stroke 1999;1(1):42~46
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