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논문 기본 정보

자료유형
학술저널
저자정보
C. L. Abayasekara (University of Peradeniya) N. K. B. Adikaram (University of Peradeniya) U. W. N. P. Wanigasekara (University of Peradeniya) B. M. R. Bandara (University of Peradeniya)
저널정보
한국식물병리학회 The Plant Pathology Journal The Plant Pathology Journal Vol.29 No.1
발행연도
2013.3
수록면
77 - 86 (10page)

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Anthracnose development by Colletotrichum musae was observed to be significantly less in the fruits of the banana cultivar ‘Embul’ (Mysore, AAB) infected with Phyllosticta musarum than in fruits without such infections. Anthracnose disease originates from quiescent C. musae infections in the immature fruit. P. musarum incites minute, scattered spots, referred to as freckles, in the superficial tissues of immature banana peel which do not expand during maturation or ripening. P. musarum does not appear to have a direct suppressive effect on C. musae as conidia of C. musae germinate on both freckled and non-freckled fruit forming quiescent infections. Our investigations have shown that P. musarum infection induced several defence responses in fruit including the accumulation of five phytoalexins, upregulation of chitinase and β-1,3-glucanase, phenylalanine ammonia lyase (PAL) activity and cell wall lignification. ¹H and <SUP>13</SUP>C NMR spectral data of one purified phytoalexin compared closely with 4"-hydroxyanigorufone. Some of the P. musarum-induced defences that retained during ripening, restrict C. musae development at the ripe stage. This paper examines the potential of P. musarum-induced defences, in the control of anthracnose, the most destructive postharvest disease in banana.

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UCI(KEPA) : I410-ECN-0101-2014-481-000296526